Neural Stem Cells to the Rescue—Can Neuroreplacement Ever Become a Treatment for Alzheimer's?
subsequent improvement of cognitive function (Qu et al., 2001) reinforces the potential feasibility of HNSC
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Vascular Disease, Parkinson's, and Now Alzheimer's—Is Homocysteine the New All-Around Bad Guy?
levels and neuropathologically pure AD (Clarke et al., 1998). That is, the homocysteine-AD link seems to
Cerebrometabolic Deficiency in Alzheimer's Disease
cholinergic mechanisms (Ragozzini et al., 1998). Gabrielle Strobel: That is interesting. I look forward to
The Neuroplasticity Theory of Alzheimer's Disease
neurofibrillary tangles. The neurofilaments appear to clog dendrite segments (Ashford et al., 1998), which leads
From Epidemiology to Therapeutic Trials with Anti-inflammatory Drugs in Alzheimer's Disease: The Role of NSAIDs and Cyclooxygenase on Beta-amyloidosis and Clinical Dementia
was the Arizona indomethacin study (Rogers et al., 1993). This was not a good study. Other therapeutic
Alzheimer Immunotherapy Trial Grounded: Time to Reassess Safety and Vaccine Design
injecting Aβ1-42 more than two years ago (Schenk et al., 1999), I was very surprised they did it in that
Hemochromatosis as a Factor in AD
evidence (Connor et al.) that the HFE protein is found on brain capillaries, choroid plexus and ependymal
Picking up the Pieces of Parkin
described in the Farrer paper (Farrer et al., 2001) is a partially active mutant as described in our Nature
The Role of ApoE in Alzheimer's Disease: An Alternative View
periphery. See (Shibata et al. 2000, Demeester et al. 2000, Rebeck et al. 1998, Koudinov et al., 2002.)
Gonadal Hormone Withdrawal, Apoptosis, and Generation of Beta Amyloid Peptide: A Vicious Cycle of Amyloidogenesis and Neurotoxicity in Alzheimer's Disease
a correlation between serum A-β levels and AD (Mehta PD et al., 2001); Vanderstichele H., 2000). Craig Atwood:
Calcium Signaling Deficit in the Origin of Alzheimer's Disease
1998 May 11;3:a27-31. Abstract. 3. Chen M. The Alzheimer's plaques, tangles and memory deficits
Nomenclature Discussion
August 8-9, 1998. Amyloid: Int J Exp Clin Invest 6, 63-66. View PubMed record.. Q. Is the failure by the
New Theories of Presenilin Function
phosphorylation and subsequent degradation of beta-catenin (Takashima et al., 1998; Zhang et al., 1998). Niwa
Generation of an Apoptotic Intracellular Peptide by γ-secretase Cleavage of Alzheimer's β-amyloid Precursor Protein
the Danish and British dementia ? Ben: BDI, Huntington's, Parkinson's, ALS, etc. So MANY
Intracellular Aβ in Alzheimer's Disease
in culture (Skovronsky et al., 1998). Recently, Aβ42 accumulation was reported both in neurons in the
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